- Type 1 is due to plaque rupture with thrombosis
- Type 2 is secondary to an imbalance between myocardial oxygen demand and supply with fixed atherosclerotic obstruction, vasospasm, or endothelial dysfunction playing a permissive role
- Type 3 includes patients with sudden death having fatal MI even though cardiac biomarker evidence is lacking.
- Types 4 and 5 include patients with MI associated with PCI and
REFERENCE: J Intensive Care Med May 2015 vol. 30 no. 4 186-200